英语翻译1 NSE染色计数神经细胞数量.2 随病程进展肺组织纤维化指数逐渐增高,神经细胞数量逐渐减少;3 CWP组逃避潜伏期较对照组延长;4 CWP组穿越平台次数较对照组大鼠减少;5 CWP组跨越目

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英语翻译1 NSE染色计数神经细胞数量.2 随病程进展肺组织纤维化指数逐渐增高,神经细胞数量逐渐减少;3 CWP组逃避潜伏期较对照组延长;4 CWP组穿越平台次数较对照组大鼠减少;5 CWP组跨越目

英语翻译1 NSE染色计数神经细胞数量.2 随病程进展肺组织纤维化指数逐渐增高,神经细胞数量逐渐减少;3 CWP组逃避潜伏期较对照组延长;4 CWP组穿越平台次数较对照组大鼠减少;5 CWP组跨越目
英语翻译
1 NSE染色计数神经细胞数量.
2 随病程进展肺组织纤维化指数逐渐增高,神经细胞数量逐渐减少;
3 CWP组逃避潜伏期较对照组延长;
4 CWP组穿越平台次数较对照组大鼠减少;
5 CWP组跨越目标象限时间占整个游泳时间的百分率较对照组明显减少;
6 煤工尘肺大鼠肺功能降低引起的慢性缺氧,可能是导致神经细胞数量减少而最终出现认知功能障碍的重要机制.
英文缩写部分不用翻译.

英语翻译1 NSE染色计数神经细胞数量.2 随病程进展肺组织纤维化指数逐渐增高,神经细胞数量逐渐减少;3 CWP组逃避潜伏期较对照组延长;4 CWP组穿越平台次数较对照组大鼠减少;5 CWP组跨越目
1 NSEStaining for counting number of nerve cells
2 With the progression of pulmonary fibrosis index increased gradually, gradually reduce the number of nerve cells;
3 Group CWP escape latency than the control group extension
4 CWPThrough the platform number group than the control group in the ratr
5 CWPGroup spans the target quadrant time accounts for the swimming time percentage decreased obviously than that in control group;
6 Coal workers' pneumoconiosis pulmonary function in rats with reduced induced by chronic hypoxia, may be the cause of the decrease in the number of neural cells resulting cognitive dysfunction are important mechanisms.

1 NSE dyeing count the number of nerve cells.2 with the progression of lung tissue fibrosis index is higher, the number of nerve cells gradually reduced;3 CWP group than controls extend from incubatio...

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1 NSE dyeing count the number of nerve cells.2 with the progression of lung tissue fibrosis index is higher, the number of nerve cells gradually reduced;3 CWP group than controls extend from incubation period;4 CWP group through a healthy rats platform times reduced;5 CWP group across goal over the entire quadrant time swimming time the percentage of a control group significantly reduced;6 coal worker's pneumoconiosis work of rats caused by chronic lung function reduce the oxygen may be contributing to the decrease in the number of nerve cells and eventually appear cognitive dysfunction important mechanism.

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